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This report summarizes work investigating the effects of some medicinal plants on uterine contraction. As there is a clinical need to find better drugs to help control uterine activity, and novel compounds are sought, the mechanisms whereby the medicinal plants exert their effects, as well as their major compounds, are discussed. By identifying the plants, major constituents and mechanisms, this review also illustrates the potential for development of new drugs, so that better ways to treat uterine disorders will be available to women worldwide. ""What is the central question of this study? [http://www.selleckchem.com/products/wnt-c59-c59.html check details] A sustained postexercise vasodilatation, which is histamine receptor mediated, has been observed following single [http://www.selleckchem.com/products/MLN8237.html selleckchem] bouts of whole-body exercise, but the mechanisms that regulate activation of histamine receptors following exercise are undefined. Exploration of vasodilatation after small muscle-mass dynamic or resistance exercise could provide novel insight into the pathways responsible for histamine receptor activation. We hypothesized that there would be a vasodilatation of the previously exercised limb following small muscle-mass dynamic and resistance exercise, which would be mediated by histamine receptors. We studied men and women before and after single-leg dynamic (n= 9) or resistance knee-extension exercise (n= 12) on control and blockade days (combined oral H1 and H2 receptor antagonism with fexofenadine and ranitidine). We measured arterial blood pressure (automated brachial oscillometry) and femoral artery blood flow (Doppler ultrasound). Dynamic exercise elevated leg vascular conductance in the active leg by 27.2 �� 8.4% at 60 min postexercise (P [https://en.wikipedia.org/wiki/Aldosterone Aldosterone] but not resistance, exercise. Furthermore, these data suggest that local factors associated with aerobic exercise, and not systemic factors or factors associated with high muscle force, are responsible for activation of histamine receptors in the previously exercised muscle. Postexercise vasodilatation of the previously active skeletal muscle vascular bed has been observed following a single bout of exercise (Halliwill, 2001). After dynamic large muscle-mass exercise (i.e. treadmill or cycle ergometry), the postexercise vasodilatation is sustained and generated by multiple mechanisms.