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		<id>http://istoriya.soippo.edu.ua/index.php?action=history&amp;feed=atom&amp;title=A_Top-secret_Knife_For_the_GSK2656157</id>
		<title>A Top-secret Knife For the GSK2656157 - Історія редагувань</title>
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		<updated>2026-04-09T00:49:20Z</updated>
		<subtitle>Історія редагувань цієї сторінки в вікі</subtitle>
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		<id>http://istoriya.soippo.edu.ua/index.php?title=A_Top-secret_Knife_For_the_GSK2656157&amp;diff=165685&amp;oldid=prev</id>
		<title>Camel2park: Створена сторінка: In contrast, fasudil treatment markedly increased the heart weights and heart index (heart [http://www.selleckchem.com/products/gsk2656157.html GSK2656157 mw] w...</title>
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				<updated>2017-04-16T10:47:46Z</updated>
		
		<summary type="html">&lt;p&gt;Створена сторінка: In contrast, fasudil treatment markedly increased the heart weights and heart index (heart [http://www.selleckchem.com/products/gsk2656157.html GSK2656157 mw] w...&lt;/p&gt;
&lt;p&gt;&lt;b&gt;Нова сторінка&lt;/b&gt;&lt;/p&gt;&lt;div&gt;In contrast, fasudil treatment markedly increased the heart weights and heart index (heart [http://www.selleckchem.com/products/gsk2656157.html GSK2656157 mw] weight/body weight) compared to the control group (Table 1). In aortic rings, the cumulative concentration�Cresponse to NE (10?9�C10?5?M) was markedly decreased in the l-NAME induced hypertensive rats compared to the control rats (P[http://en.wikipedia.org/wiki/Oxygenase Oxygenase] (P[http://www.selleckchem.com/products/i-bet151-gsk1210151a.html selleck inhibitor] Valsartan treatment did not affect this response, whereas fasudil treatment markedly enhanced the contraction of aortic segments compared to the l-NAME-evoked hypertensive group. Endothelium-dependent relaxation of aortic rings precontracted with NE was shown in Fig. 4A. Exposure to cumulative concentrations of acetylcholine (Ach) led to a marked vasorelaxation of NE-constricted rings in the control group. In contrast, the response of aortic rings from the model group to Ach was almost abolished, indicating an impaired endothelium-dependent relaxation of the aorta in the NO-deficient rats induced by l-NAME treatment. Little improvement of this effect was observed in the valsartan-treated group, but a significantly improved vasorelaxation was provided by fasudil therapy in comparison to the model group (P&lt;/div&gt;</summary>
		<author><name>Camel2park</name></author>	</entry>

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