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- We conclude that TZDs straight inhibit RANKL-induced expression of NFATc1, c-Fos and osteoclast genes, not indirectly through the suppression of RANK expression
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- We did not prospectively collect peripheral blood on these patients so it is unknown if these changes in tissue metabolic substrates correlate with peripheral abnormalities
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- We do not postulate that persistent ROS creation and oxidative pressure do not add to mtDNA harm and mutagenesis in the context of typical aging
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- We evaluated its expression degree throughout cardiac development through qRT-PCR evaluation employing isolated embryonic
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