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  15. Although both ECs and MCs showed the activation of Hes1/Hey1, it appears that the consequences of Notch activation vary in the context of stroma cell types
  16. Although irJAZd flowers did not show anthesis-related phenotypes as described in the previous section, premature flower abscission phenotype strongly resembled those of irMYB305 plants but the abscission was shifted to later stages in flower development
  17. Although it has been shown that some of these xenobiotics may alter bile acid homeostasis by inhibiting both hepatic uptake and canalicular efflux of them
  18. Although it is tempting to speculate that IL-17 may indirectly modulate p53 expression through synoviolin expression we were unable to confirm this hypothesis at least at timepoints up to 24 h of IL-17 treatment (data not shown)
  19. Although lengthy-time period Necdin overexpression was not incompatible with mobile development we cannot but conclude
  20. Although many active molecules were found by modeling interactions with this target, none of these efforts have yielded a drug that could meet regulatory approval, primarily as a result of unacceptable side effects
  21. Although the degree of backbone bending relates to (i) (Table S
  22. Although the functional relevance of PrPC compartimentation within rafts has been poorly addressed, it has recently been established that PrPC does recruit NCAM into lipid rafts
  23. Although the hemolytic action of bacterial CAMP factor has been demonstrated on erythrocytes and artificial plasma membranes
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  25. Although the subcellular localization of proteins is widely studied with many tools, its prediction is not always accurate
  26. Although these research have been performed at a later date than the previously published CV-N and MVN studies
  27. Although we identified several Ap2d- and Ash2l-regulated genes, a vast majority of differentially expressed genes did not overlap when the gene expression profiles were compared between cells treated
  28. Altogether, these results offer sturdy assist for the hypothesis that FOXO1-CA modulates activin responsiveness of the Fshb promoter by interacting with SMAD3/4 via the FOXO1 DBD
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