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  5. An additional gauge of TCR affinity makes use of the extent of peptide:MHC II tetramer staining detected by flow cytometry
  6. An additional prospective complication is that motif-pushed binding of Miz-1, this sort of as binding to Mizm1 and Mizm2
  7. An advantage of using the MSPMS assay over the PS-SCL assay for profiling biological samples is that specific cleavage of peptide substrates can be directly linked to a protease
  8. An aliquot of the same sample used for CD was analyzed by RENAGE and exhibited a bimodal size distribution with smaller oligomers (octamers to dodecamers) and larger oligomers
  9. An aliquot of total cell lysates was immunoblotted with anti eNOS antibodies to check silencing, and with anti b-actin antibodies as loading control
  10. An alternative explanation is that these cells could serve to augment the immune suppression of viral replication or could reflect a much more active antiviral response in other compartments such as lymphoid or mucosal tissue
  11. An alternative explanation is that these cells may perhaps serve to augment the immune suppression of viral replication or may perhaps reflect a far more active antiviral response in other compartments for instance lymphoid or mucosal tissue
  12. An alternative explanation is that these cells might serve to augment the immune suppression of viral replication or may well reflect a much more active antiviral response in other compartments for instance lymphoid or mucosal tissue
  13. An alternative explanation is the fact that these cells may well serve to augment the immune suppression of viral replication or could reflect a much more active antiviral response in other compartments for example lymphoid or mucosal tissue
  14. An alternative explanation is the fact that these cells may well serve to augment the immune suppression of viral replication or may reflect a extra active antiviral response in other compartments for instance lymphoid or mucosal tissue
  15. An alternative potential NLS has been suggested by others as the positively charged cluster of basic amino acids known to be critical for heparin binding
  16. An analogous modify is noticed in migrating fibroblastswhere big central adhesions are likely to disperse when MII activity is inhibited
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  18. An antagonistic method of motion on the other hand will direct to systemic outcomes in other wholesome steroidogenic tissues
  19. An best inhibitor must be extremely successful and risk-free and it ought to have steady pharmacokinetics that are only weakly
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  29. An important component of the ERAD pathway is EDEM 1, a mannosidase-like chaperone that regulates the extraction of misfolded polypeptide chains from calnexin cycle and sends them back to the cytosol for proteasomal degradation
  30. An improper peripheral accumulation of kinesin-1 may decrease the level of available kinesin-1 molecules within a cell, which could attenuate the axonal transport driven by kinesin-1
  31. An improper peripheral accumulation of kinesin-1 might reduce the volume of available kinesin-1 molecules inside a cell, which could attenuate the axonal transport driven by kinesin-1
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  33. An increase in plasma cholesterol levels was observed for most genotypes shortly after the initiation of PI regimens boosted with RTV
  34. An increased dose in RapidArc could come up from the ongoing IMRS irradiation during the total movement of tumor goal
  35. An increased dose in RapidArc may come up from the steady IMRS irradiation in the course of the whole movement of tumor goal
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  37. An intriguing discovering from our work is that in the absence of cofactor the inhibitor binds some occasions a lot more tightly than HMDP
  38. An observational and longitudinal study with retrospective data collection was designed to carry out this task, and a repeated measures model was applied for the statistical analysis, taking into account the contribution of potential confounders
  39. An option explanation is the fact that these cells may well serve to augment the immune suppression of viral replication or may possibly reflect a more active antiviral response in other compartments for instance lymphoid or mucosal tissue
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  43. An perfect inhibitor should be highly successful and safe and it ought to have stable pharmacokinetics that are only weakly
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  46. An sudden result concerns Ca2 loading among the endoplasmic reticulum and mitochondria
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