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- As E1 is existing in the diseased tissues it competes with the inhibitor for binding to the ERs
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- As a common, Western blot evaluation Western blot evaluation was performed by common procedures
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- As a negative control, anti-human body was excluded from immunoprecipitation reaction
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- As a result Necdin expression in LMPs could attenuate the response when p53 action is required
- As a result enzastaurin may exhibit antitumor activity by means of several mechanisms influencing each tumor angiogenesis and apoptosis
- As a result it would seem pertinent to hypothesize that the combination of everolimus to zoledronate could be successful in this tumor
- As a result the reduced existence of Necdin in NIHLT cells sensitized them to p53 cell cycle arrest
- As a result we centered on the discovery of compounds with out lower affinities to the ERs with no concerning agonistic
- As a typical, Western blot analysis Western blot evaluation was performed by normal methods
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- As activin treatment was associated with a decrease in p21 levels as well an increase in migration, we expected that loss of p21 by knockdown would enhance baseline migration as well as migration after activin treatment in SMAD4 intact cells
- As an alternative its C5 cysteine residue is predicted to sort a disulphide bridge with a directly adjacent
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- As an case in point be aware the correlation in between the variation in Necdin gene expression by the Affymetrix oligonucleotide microarray
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