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859, p=0.001). Males had higher BMI than the females (t=2.697, p=0.007). Mean of tTG IgA in males and females did not show any differences. Finally, BMI value for normal range (tTG IgA =20), but it was not significant (t=-1.235, p=0.217). Table 5 showed sex distribution in tTG IgA according to the cut-off point of 20. In the negative state of tTG (Selleckchem Anti-infection Compound Library for males was 95% percent. This distribution did not show any relationship between levels of tTG and sex (r=0.063, p=0.118). The confidence interval for odds ratio (0.383, 1.117) for sex factor revealed that it played the role of both risk and protection. It means that sex had no effect on tTG, but for groups, the 95% CI was equal to (0.095, 0.295) showed that belonging to each group changed the probability of having less than 20 in tTG (odds=0.285) and it means that belonging to the control group had a protective role. Also, the table showed that tTG IgA titers for CD in the case group was 11.5% (23/200), which was significantly higher than the control group with a prevalence of 3.5% (15/420)(p=0.000). Table 6 showed the correlation between BMI and tTG IgA in case and controls and combined population. The table showed that there was only a significant association in combination of population (r=0.102, p=0.011) when there was no relationship between BMI and tTG IgA in case and controls separately. Table 2 Independent t test in comparison of BMI in case and control groups Table 3 Independent t test in comparison of BMI and TTG IgA in sex groups ATP7A Table 4 Independent t test in comparison of BMI in tTG IgA status (=20) Table 5 Distribution of tTG IgA gap in sex and groups Table 6 Correlation of BMI and tTG NU7441 ic50 IgA in terms of case, control and combination Discussion The findings of our study showed a significant increase in tTG IgA in patients with Beta-thalassemia major as compared to healthy children and the tTG IgA titers for CD in the case group was 11.5% (23/200) that was significantly higher than the control group with a prevalence of 3.5% (15/420)(p=0.000). According to BMI, there was a significant difference between the two groups and growth failure was observed in the cases versus the controls, showing a significant association. There was a slightly female dominance in total participants (Male/Female=1.1/1). Ankit Parakh reported a 10- year-old boy on a hypertransfusion regimen was referred for early onset growth failure. The case had high tTG IgA level >300 IU/mL (normal value