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Completion of stage 2 of CR did not significantly affect the initial HRR60 value in the study group (16.93��8.42/min vs 17.11��7.58/min, NS) and the control group (17.20��6.50/min vs 19.10��6.71/min, NS). As a result of the CR cycle, there was a significant increment in the HRR60 value (17.98��8.33/min vs 22.72��7.72/ min, pABT-263 chemical structure Fig. 1. The ��HRR60 Rapamycin value in the study group was statistically significantly associated with the initial HRR60 value by the Pearson coefficient of ?0.595 (pFlavoprotein In turn, the heart rate recovery adaptation that takes place during the recovery phase is a result of rapid reactivation of the parasympathetic system and of a gradual disabling of the sympathetic system activity. Therefore, HRR disorders are most likely not just a result of vagus nerve disorders, but also an effect of excessive parasympathetic system activity [ 5?and?9]. Molecular mechanisms regulating the function of the parasympathetic system may also be involved in this process. It was shown that a variation in the DNA code (the acetylcholine receptor subtype M2 locus) may be responsible for the variability exhibited in HRR in healthy individuals [ 10].