Bid Apoptosis

Матеріал з HistoryPedia
Версія від 12:52, 21 червня 2017, створена Person8virgo (обговореннявнесок) (Створена сторінка: that reduce steady-state levels in the [http://www.ncbi.nlm.nih.gov/pubmed/ 23388095 23388095] transcript. J Biol Chem 283: 2827428286. 39. Cheng AM, Byrom MW,...)

(різн.) ← Попередня версія • Поточна версія (різн.) • Новіша версія → (різн.)
Перейти до: навігація, пошук

that reduce steady-state levels in the 23388095 23388095 transcript. J Biol Chem 283: 2827428286. 39. Cheng AM, Byrom MW, Shelton J, Ford LP Antisense inhibition of human miRNAs and indications for an involvement of miRNA in cell growth and apoptosis. Nucleic Acids Res 33: 12901297. 40. Wu Q, Jin H, Yang Z, Luo G, Lu Y, et al. MiR-150 promotes gastric cancer proliferation by negatively regulating the pro-apoptotic gene EGR2. Biochem Biophys Res Commun 392: 340345. 41. Urbich C, Kaluza D, Fromel T, Knau A, Bennewitz K, et al. MicroRNA27a/b controls endothelial cell repulsion and angiogenesis by targeting semaphorin 6A. Blood 119: 16071616. 42. Lerner M, Lundgren J, Akhoondi S, Jahn A, Ng HF, et al. MiRNA-27a controls FBW7/hCDC4-dependent cyclin E degradation and cell cycle get Calcipotriene progression. Cell Cycle 10: 21722183. 43. Guttilla IK, White BA Coordinate regulation of FOXO1 by miR-27a, miR-96, and miR-182 in breast cancer cells. J Biol Chem 284: 2320423216. 44. Sysa-Shah P, Xu Y, Guo X, Belmonte F, Kang B, et al. Cardiac-specific over-expression of epidermal development issue receptor two induces prosurvival pathways and hypertrophic cardiomyopathy in mice. PLoS One 7: e42805. 45. McMullen JR, Shioi T, Zhang L, Tarnavski O, Sherwood MC, et al. Phosphoinositide 3-kinase plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy. Proc Natl Acad Sci U S A 100: 1235512360. 46. Reiss K, Cheng W, Ferber A, Kajstura J, Li P, et al. Overexpression of insulin-like growth factor-1 inside the heart is coupled with myocyte proliferation in transgenic mice. Proc Natl Acad Sci U S A 93: 86308635. 47. McMullen JR, Shioi T, Huang WY, Zhang L, Tarnavski O, et al. The insulin-like development factor 1 receptor induces physiological heart growth by means of the phosphoinositide 3-kinase pathway. J Biol Chem 279: 47824793. 48. Ying H, Fu H, Rose ML, McCormack AM, Sarathchandra P, et al. Genetic or pharmaceutical blockade of phosphoinositide 3-kinase p110delta prevents chronic rejection of heart allografts. PLoS One particular 7: e32892. 49. Liang Q, De Windt LJ, Witt SA, Kimball TR, Markham BE, et al. The transcription elements GATA4 and GATA6 regulate cardiomyocyte hypertrophy in vitro and in vivo. J Biol Chem 276: 3024530253. 50. Kerkela R, Kockeritz L, Macaulay K, Zhou J, Doble BW, et al. Deletion of GSK-3beta in mice leads to hypertrophic cardiomyopathy secondary to cardiomyoblast hyperproliferation. J Clin Invest 118: 36093618. 51. Li X, Kong M, Jiang D, Qian J, Duan Q, et al. MicroRNA-150 aggravates H2O2-induced cardiac myocyte injury by down-regulating c-myb gene. Acta Biochim Biophys Sin 45: 734741. 10 ~~ ~~ Fibrin D-dimer, by far the most typically made use of clinical assay of coagulation activation and in vivo fibrin formation and lysis in circulating blood has been connected with improved risk of cardiovascular illness. A meta-analysis of prospective studies showed an association of circulating levels of fibrin D-dimer with coronary heart disease that appeared of comparable strength to that of fibrinogen. Although it has been suggested that D-dimer and other fibrin degradation solutions may perhaps have a pro-inflammatory impact, the association of D-dimer and CHD threat appears independent of inflammatory markers, for instance fibrinogen, C-reactive Protein and interleukin-6.