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However, cadmium concentration in the blood differed depending on the exposure status. Compared to participants who were never exposed to SHS, participants who were exposed to SHS longer than 1?h at work or home, and total exposure, demonstrated higher cadmium concentration (1.20 vs 1.50 with p value 0.001; 1.21 vs 1.45 with p value 0.04; 1.21 vs 1.46 with p value for a longer Selisistat supplier duration than 1?h at home and at total exposure had higher blood cadmium concentration compared with participants who were never exposed to SHS. After adjusting for confounding factors that could influence blood concentration of the metals, we confirmed that regression coefficients of linear regression models were increased with statistical significance in the analyses of cadmium concentration of total SHS exposure and SHS exposure at workplaces. (Before adjustment, regression coefficient was 0.034 with p=0.313. After adjustment, regression coefficient was 0.084 with p=0.006; before adjustment, regression coefficient was 0.042 with p=0.30. After adjustment, regression coefficient was 0.11 with p=0.005.) That means the effects of exposure to SHS on cadmium concentration in the blood became more obvious after adjusting for confounders. No significant difference was found in the levels of lead and cadmium between participants living with smokers and those not living with smokers. (For lead p=0.79 after adjustment, for cadmium p=0.43 after adjustment.) Given the general circumstances in South Korea, having cohabiting smokers does not always suggest that participants will be exposed to SHS at home, as South Korean smokers usually smoke outdoors, usually on balconies, or in gardens or public outdoor spaces near home, and not indoors. Therefore, despite having cohabitants who are active smokers, participants might not be exposed to a significant amount of SHS in South Korea. Several previous studies support the results of this work. Tobacco is well known as a notable source of cadmium and lead.