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3-fold, p?tiospirone with combined ABCA1 and SR-BI deficiency, a necropsy of the mice at the age of 16 weeks old on chow diet was performed. No significant differences were observed in liver weight between WT, ABCA1 KO, SR-BI KO, and dKO mice (50.0?��?1.8?mg/g, 55.3?��?2.1?mg/g, 56.0?��?3.0?mg/g, 52.5?��?3.3?mg/g body weight, respectively). In line with previous findings [8], [25]?and?[26], ABCA1 KO mice and SR-BI KO mice showed a 1.3-fold (4.4?��?0.2?mg/g, p?17-AAG supplier body weight (p?Docetaxel cell line accumulation. Strikingly, dKO mice displayed more extreme macrophage foam cell formation in the lung and Peyer's patches as evidenced by colocalization of Oil-red-O with Moma-2 macrophage staining (Fig. 6A). Lipid quantification indicated that accumulated neutral lipid in the lung is cholesterol ester (dKO: 204.2?��?31.04?��g/mg protein vs WT: 1.8?��?1.2?��g/mg protein, p?