Внесок користувача
- 13:42, 21 січня 2017 (різн. • історія) . . (+3769) . . Н These studies implicate that GRKs, especially GRK5, may exert multiple physiological functions via various mechanisms including those independent (Створена сторінка: Numerous research show that GRKs are capable to phosphorylate a selection of non-GPCR substrates this kind of as synuclein [three], p38 [four], NF-kB1 p105 [5],...) (поточна)
- 08:41, 21 січня 2017 (різн. • історія) . . (+3901) . . Н This data is consistent with our in vitro findings of TGFb/SMAD4dependent upregulation of p21 and activin/non-SMAD4-dependent downregulation of p21 (Створена сторінка: GAPDH was employed as loading management and relative expression was calculated via densitometry. D) Even though TGFb-induced upregulation of p21 was SMAD4 dep...) (поточна)
- 21:40, 19 січня 2017 (різн. • історія) . . (-11) . . м ACVR2/TGFBR2/SMAD4-wild type FET cells were were pretreated for 30 minutes with proteasomal inhibitor MG-132 and then treated with vehicle (control) (поточна)
- 20:31, 19 січня 2017 (різн. • історія) . . (+4140) . . Н ACVR2/TGFBR2/SMAD4-wild type FET cells were were pretreated for 30 minutes with proteasomal inhibitor MG-132 and then treated with vehicle (control) (Створена сторінка: The decline of the two of these signaling pathways may possibly be useful and additive for tumor expansion [twenty,27], but the differential impact on migratio...)
- 12:57, 19 січня 2017 (різн. • історія) . . (+3615) . . Н As activin treatment was associated with a decrease in p21 levels as well an increase in migration, we expected that loss of p21 by knockdown would enhance baseline migration as well as migration after activin treatment in SMAD4 intact cells (Створена сторінка: As [http://www.medchemexpress.com/APD597.html 897732-93-3] activin remedy was associated with a lower in p21 levels as properly an enhance in migration, we ant...) (поточна)
- 12:25, 17 січня 2017 (різн. • історія) . . (+3898) . . Н The modulation of the Th1/Th2 balance can induce either the death or proliferation of intracellular Leishmania in the macrophages (Створена сторінка: The modulation of the Th1/Th2 balance can induce both the loss of life or proliferation of intracellular Leishmania in the macrophages [sixty eight], and L-argi...) (поточна)