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15 Our results were much in the same vein. While the progression or regression of the fracture displacement varied somewhat, we found no statistically significant difference between zone I and zone II sacral fractures in either MAPK inhibitor final fracture displacement or average change in fracture displacement from the postoperative period to final follow up, confirming the role of stable early fixation. Our data also showed that the change of fracture displacement from the postoperative period to final follow up was significant (zone II P?=?0.03, overall cohort P?=?0.02). In both the overall cohort and patients with zone II fractures, anterior stabilization of the pelvic ring with plate and screws yielded positive numbers, Telomerase indicating a decrease in displacement at the fracture site. Conversely, the patients in these two groups who had either external fixators placed or no fixation of their anterior ring had negative average changes in their fracture displacement, indicating that the fracture displacement increased from the postoperative period to the final follow-up period. Normally described as displacement of >1?cm at final follow up, failure of the mode of stabilization is the focus of much of the published literature on anterior fixation used with posterior stabilization. We found that the mode of anterior fixation played no statistically significant role in how much displacement was noted on the final follow-up radiographs. This would suggest that the instability in zone II fractures may be implicated in the displacement observed over time at the fracture site, while the final displacements in the overall cohorts were not significantly different between the different zones or by the different modes of anterior fixation. A review of the mean values may suggest a trend towards statistical significance when some comparisons are made; examples include zone I ventral displacement of 6.8?mm and 3.36?mm with an external fixator and anterior ring fixation, respectively. This trend was slightly reversed in zone II fractures, where external fixation was associated with follow-up ventral displacement of 5.25?mm, and no fixation was associated with displacement of 7.43?mm. Of note, RC displacement is controlled by the posterior fixation, which Androgen Receptor antagonist mimics the innate mode of stabilization: posterior SI ligaments. All patients in all cohorts averaged