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All environmental exposures differed significantly between rural and urban households (Table 2). Rural households had higher exposure to livestock, visible excrement around the house, particulate-generating cooking fuel, and car/motorcycle exhaust. Conversely, find more urban households had higher incomes and a greater proportion had access to running water and flushing toilets. The prevalence of moderate (1�C10mg/L) and acute (>10 mg/L) CRP levels in children was 7.89% and 2.77%, respectively. Adults exhibited a higher prevalence of moderate inflammation (CRP 3�C10mg/L) at 21.42% and acute inflammation (CRP>10mg/L) at 3.58%. The prevalence of acute CRP was significantly higher in adults with low household income compared to medium/high income (4.11% vs 3.18%; p CRP by urban/rural residence in either children or adults (Fig. 2). We found statistically significant associations between individuals' illness histories and elevated CRP in the initial individual models (Table 3). Few symptoms were associated with the risk of elevated CRP in children; however, children and adolescents with a history of diarrhea in the past month were more likely selleck products to have moderately elevated CRP levels than normal CRP (RRR: 3.52; 95% CI 1.01, 12.27). The associations between symptom history and inflammation were more extensive in adults in whom both infectious and non-communicable illness symptoms were associated with an increased likelihood of having elevated CRP. Experiencing any of the reported symptoms, with the exception of asthma symptoms which did not reach the pCasein kinase 2 symptoms, fever/cough/sore throat, or asthma in the past month was associated with an increased likelihood of acutely elevated CRP. Thus, in adults, both infectious and non-communicable symptoms were associated with moderately elevated CRP while infectious symptoms and asthma were associated with acutely elevated CRP. Household environmental exposures were significantly associated with the risk of elevated CRP in adults but not children in initial models (Table 4). In children and adolescents, only exposure to particulate-producing cooking fuels was associated with a higher risk of moderately elevated CRP, but this association did not reach a p