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This paper presents the findings and views of its author and not necessarily those of the US Environmental Protection Agency. The author currently provides consulting services Selleckchem Capmatinib on indoor air quality matters under DM Indoor Air Consulting, and the author is a part time employee at The Cadmus Group Inc., which is a consulting group. Competing Interests The author declares no competing interests.""Statistical tests were performed using STATISTICA for Windows Version 10.0 (StatSoft Inc. Europe, Hamburg, Germany). The chi squared test was performed to compare parameters of the analysed subcohort with the remaining LINA cohort (N: 546 ? 68 = 478). Analyses related to Eo/B CFUs are calculated within the described subcohort (N = 68 mother-child pairs, including two sets of twins). Calculations for general associations independent of Eo/B CFU analyses (e.g., ETS exposure versus VOC concentrations) were performed for the entire LINA cohort (N = 546). When statistical analyses required a division into subgroups (e.g., exposure to ETS: yes versus no) data were not presented for selleck products N-numbers Quinapyramine analyses were performed using nonparametric tests in general. To address the relationship between the numbers of Eo/B CFUs and atopic outcomes or exposure to indoor ETS or disinfectants, medians were compared using the Mann-Whitney U test. To verify these results, multiple logistic regression models were used to determine adjusted odds ratios (OR) with a 95% confidence interval. The following confounders were used: month of birth (for seasonal changes), gender of the child (for potential differences between boys and girls), parental school education (as a marker for the social status), and family history of atopy (to consider the individual genetic background), as well as exposure to indoor ETS during pregnancy, maternal cotinine level at the child's first birthday, the sum of all measured VOCs at the child's first birthday (both cotinine and VOCs as a marker for ETS exposure in the second year of life), dampness in the dwelling during second year of life (as a marker for early airway allergen exposure), and children with infections of the airways in the second year of life (to consider a potential nonallergic origin of wheezing).