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- This effect could hardly be ascribed to variations in electrophilicity suggesting these substituents
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- This enables substrate proteins to enter the catalytic website for phosphorylation aggressive inhibitors
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- This holds also accurate for the widespread illnesses with inflammatory and autoimmune pathologies these kinds of as rheumatoid arthritis
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- This is achieved mainly by inducing the differentiation and recruitment of smooth muscle cells and pericytes
- This is followed by the generation of open complex formation (Figure 7, upper half), so that other components of DNA replication can easily be loaded
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- This is more simply witnessed by normalizing values to the suitable handle so that the thrombin-dependent reaction is distinct (Fig. 6B)
- This is not pertinent in terms of the pursued therapeutic concept which aims at excluding systemic results
