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- This capability is mediated principally by means of the binding immortalization of mouse primary cells in culture
- This capacity is mediated principally through the binding immortalization of mouse main cells in lifestyle
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- This effect could hardly be ascribed to variations in electrophilicity suggesting these substituents
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- This effect was most prominent in case of ciproxifan (12), but also pronounced for 19 and 20
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- This enables substrate proteins to enter the catalytic website for phosphorylation aggressive inhibitors
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- This holds also accurate for the widespread diseases with inflammatory and autoimmune pathologies such as rheumatoid arthritis
- This holds also accurate for the widespread illnesses with inflammatory and autoimmune pathologies these kinds of as rheumatoid arthritis
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- This indicated that induction of Hey gene expression was unbiased of Notch receptor cleavage. We recurring this experiment with a different variety of Notch inhibitor
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- This is a tiny sample but consists of all the genes that demonstrate the greatest transcriptional
- This is achieved mainly by inducing the differentiation and recruitment of smooth muscle cells and pericytes
- This is followed by the generation of open complex formation (Figure 7, upper half), so that other components of DNA replication can easily be loaded
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- This is more simply witnessed by normalizing values to the suitable handle so that the thrombin-dependent reaction is distinct (Fig. 6B)
- This is not pertinent in terms of the pursued therapeutic concept which aims at excluding systemic results
- This is not related in terms of the pursued therapeutic notion which aims at excluding systemic effects
- This is particularly true for PDE5A and PDE1B whose kinetic parameters regarding MANT-cGMP are close to the values for native cGMP
- This is proved by regimen clinic use of different endocrine therapies for instance with GnRH analogues
- This is simply because the influence of respiratory motion can't be perfectly simulated in the dose calculation process
- This is supported by other recent studies that show that damage can also lead to nonlethal autophagy
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- This is the very first demonstration that iPLA2c has a earlier unrecognized homeostatic position in a specific lineage of hematopoietic cells, particularly platelets
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- This kind of a method has been properly utilized to overcome resistance to b-lactams because of to b-lactamase exercise
- This kind of a method has been successfully used to overcome resistance to b-lactams owing to b-lactamase exercise
- This kind of a therapeutic approach has already been revealed to be effective in androgen dependent diseases like benign prostate hyperplasia
- This kind of a therapeutic strategy has already been shown to be effective in androgen dependent illnesses
- This kind of autoantigens could trigger immune responses in the two the human male and female reproductive systems
- This kind of small scaffolds led to the identification of forty-three compounds between the noted TB actives
- This latter exon is flanked by introns in Aotra and Cctra. Both Botra and Aotra have an additional male-specific exon, ms3
- This latter technique revealed that agglomerated CaP particles settled rapidly on VSMCs, and that the cells consequently retracted and produced plasma membrane blebs that gradually expanded and protruded from the cell surface
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- This made it difficult to unequivocally identify the endocytic compartments harboring NPs within the IEC cytoplasm
- This may be because a1A-AR activation in vivo is not sustained but intermittent, fluctuating with endogenous catecholamine levels
- This may be due, in part, to the dynamic and unstable constitution of this modification, and to the difficulty of the purification of O-GlcNAcylated proteins
- This may be due to the incomplete depletion of MP2, since the knockdown efficiency of MP2 in transgenetic flies
- This may be due to the short half-life of bone morphogenic proteins, but also due to the fact that Hey1 and Hey2 proteins repress the expression of their own gene promoter
